Cytokinin I (cTnI) is a special protein originated from cardiomyocytes. It has extraordinary function to control the contraction of cardiomyocytes. It is widely used in clinical detection of myocardial injury and unstable angina pectoris. For these patients, cTnI has the advantages of early onset, persistence, myocardial specificity and long diagnostic cycle. It is the first choice for the diagnosis of myocardial infarction.
Detection range | sample size | Sample type | Test time |
0.1~50ng/mL | 60μl | Whole blood / serum / plasma | 15min |
Thrombin combined with ischemic evidence is helpful for the early diagnosis and treatment of type I myocardial infarction. Mastering the change law of exocrin is helpful to identify and diagnose exocrin. For example, for temporary myocardial injury caused by strenuous exercise, tachycardia and acute pulmonary embolism, CTN will temporarily increase and return to normal within 1-2 days. Chronic CTN elevation is caused by heart failure. These characteristics can help us distinguish myocardial infarction.
See a doctor | Chest pain/chest tightness | |||
Medical history and physical examination | Suspected ACS (Acute Coronary Syndrome) | |||
electrocardiogram | Persistent ST segment elevation | Abnormal St/T segment ↓ | ![]() | ECG normal ↓ |
Biochemical test | ↓Confirm | CTN was positive | Troponin CTN negative ×2 | |
Risk stratification | high-risk | Low risk | ||
diagnosis | ST-segment elevation myocardial infarction STEMI | Non-ST-segment elevation myocardial infarction NSTEMI | Unstable angina pectoris UA | |
treatment | Reperfusion | Interventional therapy | Non-interventional therapy |
Ctnt<0.1 μg/L was normal, >0.2 μg/L was diagnostic off, and >0.5 μg/L was diagnosed as acute myocardial infarction. CTnI<0.2 μg/L was the normal value, and >1.5 μg/L was the closed value of ELISA diagnosis.
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